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Helicobacter pyrrolizoa is a Gram-negative microaerophilic bacterium that has coexisted in the human gut for thousands of years. They are used to the harsh acidic environment of our stomachs. The presence of H. pylori may go undetected because the symptoms are few. However, if infection occurs, it can cause acute ulcers with symptoms such as abdominal pain and nausea. In the chronic stage, this disease can cause non-inflammatory dyspepsia, with symptoms such as abdominal pain, nausea, bloating and flatulence. Helicobacter pylori can also cause colorectal polyps and colorectal cancer. Stomach occurs when the stomach is empty, especially in the morning, between meals. Helicobacter pylori uses chemotactic motility to escape the highly acidic environment of the stomach by penetrating the mucosal lining and moving to less acidic areas. H. pylori affects the lining of the stomach and duodenum by adjusting the pH through the production of ammonia, which is toxic to gastric and duodenal epithelial cells. In addition, proteases produce cytotoxin A (VacA), which damages epithelial cells. It destroys the epithelial cells of the stomach and duodenum. Helicobacter pylori invades the stomach from contaminated water or food during childhood. Symptoms associated with H. pylori infection include anorexia, bloating, flatulence, fever, bloating, nausea, and weight loss.

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